ISIS Report 13/06/12Syngenta Charged for Covering up Livestock Deaths from GM Corn
Corporation faces criminal charges for concealing own study in which cows died after eating its genetically modified corn Dr Eva Sirinathsinghji http://www.i-sis.org.uk/
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Biotech
giant Syngenta has been criminally charged with denying knowledge that
its genetically modified (GM) Bt corn kills livestock during a civil
court case that ended in 2007 [1].
Syngenta’s Bt 176 corn variety expresses an insecticidal Bt toxin (Cry1Ab) derived from the bacterium, Bacillus thuringiensis (Bt)
and a gene conferring resistance to
glufosinate herbicides. EU cultivation of Bt 176 was discontinued in
2007. Similar varieties however, including Bt 11 sweet corn are
currently cultivated for human and animal consumption in the EU.
The
charges follow a long struggle for justice by a German farmer whose
dairy cattle suffered mysterious illnesses and deaths after eating Bt
176. They were grown on his farm as part of authorised field tests
during 1997 to 2002. By 2000, his cows were fed exclusively on Bt 176,
and soon illnesses started to emerge. He was paid 40 000 euros by
Syngenta as partial compensation for 5 dead cows, decreased milk yields,
and vet costs (see [2] Cows ate GM Maize and Died, SiS 21).
During a civil lawsuit brought against the
company by the farmer however, Syngenta refused to admit that its GM
corn was the cause, claiming no knowledge of harm. The case was
dismissed and Gloeckner remained thousands of euros in debt.
Gloeckner
continued to lose cows and many more had to be put down due to serious
illnesses, compelling him to stop using GM feed from 2002. He approached
the Robert Koch Institute and Syngenta to conduct a full investigation.
However, only one cow was ever analysed and the data are still
unavailable to the public. Unsurprisingly, no causal relationship
between the GM feed and deaths was determined; and there is still no
explanation for the deaths.
But
in 2009, the farmer learned of a feeding study allegedly commissioned
by Syngenta in 1996 that
resulted in four cows dying in two days. The trial was abruptly
terminated. Now Gloeckner, along with a German group called Bündnis
Aktion Gen-Klage and another farmer turned activist Urs Hans, have
brought Syngenta to the criminal court to face charges of withholding
knowledge of the US trial, which makes the company liable for the
destruction of the farmer’s 65 cows. Syngenta is also charged with the
deaths of cattle in the US trial and on Gloeckner’s farm, which should
have been registered as “unexpected occurrences”. Most seriously, the
German head of Syngenta Hans-Theo Jahmann, is charged for withholding
knowledge of the US study from the judge and from Gloecker in the
original civil court case.
Gloecker’s cows not alone
This
is by no means the only account of mysterious deaths associated with Bt
GM feed. In India where livestock are left to graze on post-harvest
cotton, thousands of livestock deaths have been recorded in different
villages across central India where Bt cotton is grown (see [3] Mass Deaths of Sheep Grazing on Bt Cotton, SiS 30).
Shepherds’ own observations and post-mortem analysis carried out in the
laboratory revealed abnormal liver, enlarged bile ducts and black
patches in the intestine. The shepherds said that the sheep became
“dull/depressed” after 2-3 days of grazing, started coughing with nasal
discharge and developed red lesions in the mouth, became bloated and
suffered blackish diarrhoea, and sometimes
passed red urine. Death occurred within 5-7 days of grazing. Sheep from
young lambs to adults of 1.5-2 years were affected. One shepherd
reported getting diarrhoea from eating the meat of an affected sheep.
The vets declared that the toxicity could be due to the Bt toxin but
this could not be proven as results were confounded by additional
pesticides used on the fields. The shepherds were however, advised
against letting the sheep graze on any more Bt cotton plants.
Philippine
villagers living around Bt Maize fields have also suffered deaths and
similar illnesses of fever, respiratory, intestinal and skin problem
(see [4] GM ban long overdue, five deaths and dozens ill in the Philippines, SiS 29).
Five mortalities were reported in 2003 and subsequently, 38
individuals had their blood analysed and all were positive for
antibodies specific to Cry1Ab, suggesting an immune reaction to the
toxin. As is often the case, intimidation and denial by government
officials meant that there were no further investigations into the
matter.
Cause of deaths unknown
There
is still no explanation provided by the authorities as to the cause of
death of Gloeckner’s cows. The biotech industry claims that Bt toxins
are quickly digested in the stomach and are only effective in insect
target species. However, a recent study has found the toxin in the blood
of over 80 %
of women and their unborn children tested in Canada [5]. Because
naturally existing Bt toxins from the soil bacterium have been used for a
long time, long-term toxicology and health risk assessments on Bt
proteins in GM crops were not done. However, there are important
differences between the naturally produced toxins that can be washed off
the crops, as opposed to genetically modified toxins that are part and
parcel of the GM crop. Independent studies have shown that basing health
assessments on flawed scientific assumptions is not only arrogant, but
foolish.
Scientific
studies dating from the 1990s have identified Bt toxins as potent
immunogens, with Cry1Ac inducing immune responses in mice similar to the
cholera toxin [6]. Farm workers dealing with Bt cotton have
consistently reported allergic responses requiring hospitalisation in
some cases (see
[7] More Illnesses Linked to Bt Crops, SiS 30).
Binding of Cry1Ac to the intestine of mice has been shown, with
concomitant diarrhoea symptoms [8]. A meta-analysis of 3 month feeding
studies in laboratory animals found that Bt maize led to changes in
blood protein levels indicative of abnormal liver metabolism (see [9] GM Feed Toxic, Meta-Analysis Confirms, SiS 52). A recent study finds Cry1Ab toxic to human kidney cells, causing cell death at low doses (see [10] Bt Toxin Kills Human Kidney
Cells, SiS52).
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